Nothing beyond anecdotes, a lot of which are coming from social media and therefore notoriously unreliable. For example this message which has been widely circulated online regarding "4 young people in a serious condition" who were taking NSAIDs, which was called out as being fake by the society who supposedly released it - https://www.thejournal.ie/ibuprofen-cuh-coronavirus-whatsapp-5047311-Mar2020/

The underlying mechanism being proposed is that NSAIDs and ACE inhibitors cause up-regulation of the ACE2 receptor, and it is this receptor which allows SARS-CoV-2 gain entry to human cells. However, there is also evidence that ACE2 exhibits a protective effect in ARDS (acute respiratory distress syndrome), which is what patients with COVID-19 are dying from. The position statement from the European Society of Cardiology is to continue taking anti-hypertensive therapy as prescribed by your doctor, which I would agree with. However, unless you have a strong reason to be taking a NSAID (like rheumatoid arthritis, for example), I would probably be avoiding them and sticking to acetaminophen/paracetamol.

Having said all that, it makes little difference to my individual practice in intensive care. I couldn't tell you the last time I used a NSAID in intensive care, and the vast majority of our patients with severe infections, sepsis, multi-organ failure, etc. are hypotensive, rather than hypertensive, and generally have most, if not all, of their anti-hypertensive medication stopped during their time with us.